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. 2019 Jul 29;17:86. doi: 10.1186/s12964-019-0393-8

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — HDAC inhibition decreased doxorubicin resistance of HL-60/ADM, K562/A02 and primary BMCs in refractory AML. a doxorubicin-releasing index of AML cells after treatment with AR-42, an inhibitor of HDACs. b doxorubicin-releasing index of AML cells after treatment with Panobinostat, another inhibitor of HDACs. c doxorubicin-releasing index of AML cells after treatment with SNDH-275, a specific inhibitor of HDAC1. d MRP1 protein expression was detected by western blot. Data are the mean ± SD of three independent experiments in AML cells. ^**P < 0.01 vs. control