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. 2009 Oct 21;29(42):13283–13291. doi: 10.1523/JNEUROSCI.3069-09.2009

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Copyright © 2009 Society for Neuroscience 0270-6474/09/2913283-09$15.00/0

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Figure 6. — NO donor DEA/NO increases the locomotor frequency via depression of midcycle inhibition. A, Intracellular recording from a MN that received phasic on-cycle excitation with the ipsilateral ventral root (vr-i) and midcycle inhibition in phase with the contralateral ventral root (vr-c). B, Application of DEA/NO (100–200 μm) increased the locomotor frequency in parallel with a depression of the midcycle inhibition. C, Averaged synaptic inputs received by the MN. The action potentials were filtered out using a low pass filter. DEA/NO decreased the amplitude of the midcycle inhibition, while the on-phase excitation was not decreased. D, Averaged data from all experiments showing the time course of the NO-mediated increase of the locomotor frequency (p < 0.001, n = 8) is in parallel with a decrease in the midcycle inhibition (p < 0.0001, n = 8).