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. 2019 Jul 31;9:11087. doi: 10.1038/s41598-019-47597-4

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Deprivation of p62 reverses the inhibition of ST-induced autophagy. 786-O (a), ACHN (b) and HeLa (c) cells were transfected with the p62 siRNAs (sc-29679) for 48 h. HEK293T cells with either wild type (WT) or mutated p62 plasmid (d). The lysates were analyzed by immunoblotting (8–13% PAGE) following ST (8 μM/L or as indicated) treatment for 4 h in the presence or absence of CQ (ACHN: 10 μM/L; HeLa: 15 μM/L; 786-O: 20 μM/L; HEK293T 20 μM/L). Actin was used as the loading control. The results were similar among experiments repeated three times. (L-Exp: long exposure; Actin: A).