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. 2019 Jul 31;10:3421. doi: 10.1038/s41467-019-11385-5

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — TEs exhibit strong and diverse chromatin-associated insertion biases towards genes. a Circos representation of private TE insertions detected for VANDAL21, ATENSPM3, and ATCOPIA93 within wild-type intervals. Centromere positions are indicated by black dots. b Fraction of TE insertions in genes, TEs and intergenic regions. c Fraction of essential genes among genes targeted by VANDAL21, ATENSPM3 or ATCOPIA93 in the epiRILs. Statistical significance for each comparison was obtained using the Chi-square test. d Metagene analysis showing the distribution of new insertions. e Relative abundance of insertion sites in relation to the nine chromatin states defined in A. thaliana. f Levels of DNAse hypersensitivity (DH; top panel), H3K7me3 (middle panel), and H2A.Z (bottom panel) around VANDAL21, ATENSPM3, and ATCOPIA93 insertion sites, respectively. Source data are provided as a Source Data file