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. 2019 Aug 1;11:67. doi: 10.1186/s13195-019-0522-z

Table 1.

Comprehensive overview of end point immunohistochemistry and longitudinal FDG-PET findings in treated and untreated hTau mice

hTau Anle138b hTau Vehicle hTau pooled Baseline Non-carrier controls (pooled)

Immunohistochemistry

Frontal cortex (%-area)

6.1 ± 1.9** 12.9 ± 1.8 0

Immunohistochemistry

Frontal cortex (CP13+ neurons, N/mm2)

1366 ± 116** 2039 ± 153 0

Immunohistochemistry

Hippocampus (%-area)

1.9 ± 1.4** 4.8 ± 0.4 0

Immunohistochemistry

Hippocampus (CP13+ neurons, N/mm2)

534 ± 18** 619 ± 13 0

FDG-PET baseline

Frontal cortex (SUVR)

0.97 ± 0.02 1.02 ± 0.09 1.00 ± 0.07# 1.10 ± 0.10

FDG-PET follow-up

Frontal cortex (SUVR)

1.03 ± 0.05* 0.95 ± 0.03# 1.09 ± 0.06

FDG-PET change

Frontal cortex (ΔSUVR)

+ 0.06 ± 0.06* − 0.07 ± 0.09 + 0.01 ± 0.05

FDG-PET baseline

Hippocampus (SUVR)

0.93 ± 0.03 0.98 ± 0.10 0.96 ± 0.08# 1.06 ± 0.10

FDG-PET follow-up

Hippocampus (SUVR)

1.00 ± 0.07 0.93 ± 0.05## 1.05 ± 0.07

FDG-PET change

Hippocampus (ΔSUVR)

+ 0.06 ± 0.05 − 0.05 ± 0.10 + 0.00 ± 0.05

Results summary: “*” indicates p < 0.05 and “**” indicates p < 0.005 in the direct comparison of hTau treatment and vehicle groups. “#” indicates p < 0.05 and “##” indicates p < 0.005 in the comparison of hTau groups (pooled baseline or treatment/vehicle during follow-up) versus non-carrier controls