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. 2007 Apr 18;27(16):4482–4491. doi: 10.1523/JNEUROSCI.0680-07.2007

Figure 6.

Figure 6.

GR antagonist prevents the increased intrinsic excitability and positive shift in GABAA reversal potential produced by CORT (100 nm). A, Scheme of the protocol used in this experiment. The GR antagonist RU38486 (5 μm) was added to the ACSF before, during, and after the CORT (100 nm) application. B, Graph plotting the normalized change in, from left to right, Vm, Rin, GABAA reversal potential, and proportion of SA BLA neurons in slices exposed to CORT alone (red bars; 100%) versus CORT and RU38486 (gray bars). RU38486 prevented all of the effects of CORT (100 nm), with the exception of the shift in resting potential.