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The proposed model for the ATP‐hypoxia‐inducible factor (HIF) signaling‐2α axis in breast cancer invasion and the epithelial‐mesenchymal transition (EMT) process. ATP could upregulate HIF‐2α via AKT‐PGK1 signaling, provoking HIF‐2α targets, among which LOXL2 and MMP‐9 mediate ATP‐driven invasion, and E‐cadherin and Snail mediate ATP‐driven EMT
