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. 2019 Jul 12;18(3):1752–1760. doi: 10.3892/etm.2019.7769

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Copyright: © Gong et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 4. — Effects of ox-LDL and PRR gene silencing on oxidative stress levels. (A) LDH and (B) H₂O₂ concentrations were detected using chemical colorimetry. (C) ROS levels were tested by flow cytometry. (D) Flow cytometry results for the different groups are shown. *P<0.05 and **P<0.01, vs. control group; ^##P<0.01, vs. ox-LDL group; ^{^^}P<0.01, vs. ox-LDL + mock groups; ^&P<0.05 and ^&&P<0.01, vs. ox-LDL + siPRR group. ox-LDL, oxidized low-density lipoprotein; PRR, renin/prorenin receptor; siPRR, small interfering RNA-PRR; LDH, lactate dehydrogenase; H₂O₂, hydrogen peroxide; ROS, reactive oxygen species.