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. 2019 May 21;72(8):542–549. doi: 10.1136/jclinpath-2019-205818

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© Author(s) (or their employer(s)) 2019. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ.

This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited, appropriate credit is given, any changes made indicated, and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/.

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Correlations between p16 status, programmed death ligand 1 expression on tumour cells (TC-PD-L1), programmed death ligand 1 expression on immune cells (IC-PD-L1) and CD8⁺ tumour-infiltrated lymphocyte density (TIL). (A) TC-PD-L1 expression level according to p16 status. (B) IC-PD-L1 expression level according to p16 status. (C) CD8⁺ TIL density according to p16 status. (D) TC-PD-L1 expression level according to high or low CD8⁺ TIL density. (E) Tumour immune microenvironment types classified by TC-PD-L1 level and CD8⁺ TIL density according to p16 status. Significant differences were evaluated using the Wilcoxon and the Fisher’s exact tests.