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. 2019 Jul 18;8(7):741. doi: 10.3390/cells8070741

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© 2019 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Pathomechanism of HCM and key PQC players. (a) Stabilizing HSPs (HSPB1, HSPB5, HSPB7), (b) HSPs with refolding capacity (HSPD1, HSPA1 HSPA2) and (c) degradation markers (ubiquitin, p62, LC3BII) were not significantly different between HCM_HI (n = 19) and HCM_PP (n = 19). Each dot in the scatter plots represents an individual sample. Figure reproduced and adapted with permission from Dorsch, L.M. et al., Pflügers Archiv–European Journal of Physiology; published by Springer Berlin Heidelberg, 2018.