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. 2019 Jul 30;10:955. doi: 10.3389/fphys.2019.00955

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Copyright © 2019 Stolp, Fleiss, Arai, Supramaniam, Vontell, Birtles, Yates, Baburamani, Thornton, Rutherford, Edwards and Gressens.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Developmental pattern of interneuron injury changes by P10 in mice with inflammation-induced brain damage. Interneuron populations were assessed again at P10 by immunohistochemistry (A) and quantified (B–E) for number and distribution through cortical layers. There was a significant decrease in the number of parvalbumin (PV)-positive interneurons across the cortex (B; treatment effect, p = 0.01, two-way ANOVA), but no change for somatostatin (SST), neuropeptide Y (NPY), calretinin (CalR), or calbindin (CalB, C–F). Data presented as mean ± SD; scale bar = 100 μm. ^*p < 0.05.