Skip to main content
. 2019 Aug 2;12:6175–6180. doi: 10.2147/OTT.S207079

Figure 4.

Figure 4

TGF-β1 promoted GAPLINC expression to accelerate  non-small cell lung cancer (NSCLC) cell migration and invasion. Analysis of transwell migration and invasion showed that TGF-β1 and GAPLINC overexpression caused the accelerated migration (A) and invasion (B) of H23 and H522 cells. In addition, silencing of GAPLINC siRNA attenuated the effect of TGF-β1 treatment (*p<0.05).