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. 2019 Aug 7;9:11488. doi: 10.1038/s41598-019-47865-3

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Plasmodium berghei Anka infection during pregnancy elicited an intense maternal inflammatory response. (A) Chart of the experimental design. Measurement of specific cytokine and chemokine contents in the maternal serum from pregnant mice infected with Plasmodium berghei ANKA on GD18.5. (B) IL1-β (n = 10/8); (C) IL-6 (n = 9/10); (D) CXCL1 (n = 10/10); (E) MCP-1 (n = 10/10). Values are expressed as the mean ± standard error. The unpaired Student’s t test was employed the for IL1-β, IL-6 and MCP-1 comparisons, and the Mann-Whitney test was used for comparing CXCL1 values.