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. 2019 Jul 15;8(8):1159–1167. doi: 10.1530/EC-19-0243

Table 3.

Correlation between serum concentration of fetuin-B with anthropometric and biochemical parameters in PCOS and control group.

PCOS (n = 57) Control (n = 51)
BMI r = 0.32, P = 0.01 r = 0.33, P = 0.01
Waist circumference r = 0.30, P = 0.02 r = 0.19, P = 0.16
Fat mass (%) r = 0.27, P = 0.04 r = 0.12, P = 0.39
Fat free mass (kg) r = 0.14, P = 0.30 r = 0.30, P = 0.02
Baseline insulin r = 0.36, P = 0.007 r = 0.09, P = 0.6
HOMA-IR r = 0.31, P = 0.01 r = 0.07, P = 0.6
HOMA-B r = 0.43, P = 0.01 r = 0.66, P = 0.06
TG r = 0.30, P = 0.02 r = 0.22, P = 0.12
FAI r = 0.26, P = 0.04 r = 0.33, P = 0.01
FLI r = 0.29, P = 0.02 r = 0.24, P = 0.08
VAI r = 0.29, P = 0.02 r = 0.21, P = 0.13
LAP r = 0.32, P = 0.01 r = 0.25, P = 0.69

Data are derived from Spearman’s rank correlation. The level of significance was accepted at P < 0.05.

FAI, free androgen index; FLI, fatty liver index; HOMA-B, homeostasis model assessment β cell function; HOMA-IR, homeostasis model assessment of insulin resistance; LAP, lipid accumulation product; PCOS, polycystic ovary syndrome; TG, triglycerides; VAI, visceral adiposity index.