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. 2019 Jul 22;74(3):687–696. doi: 10.1161/HYPERTENSIONAHA.118.12409

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© 2019 The Authors.

Hypertension is published on behalf of the American Heart Association, Inc., by Wolters Kluwer Health, Inc. This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution, and reproduction in any medium, provided that the original work is properly cited.

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Figure 2. — Cardiac phenotype in spontaneously hypertensive rat (SHR) and Camk2n1^−/− rats. A, Heart and (B) left ventricle (LV) wet masses. C, Rate pressure product (RPP). D, LV CaMKII (Ca²⁺/calmodulin-dependent kinase II) activity. Average transcript levels normalized to hypoxanthine-guanine phosphoribosyltransferase, Hprt for (E) α-skeletal actin, Acta, α-cardiac actin, Actc, the Acta:Actc ratio and (F) natriuretic protein b, Nppb. Mean±SEM n=5–7/group. Significant differences by genotype (SHR and Camk2n1^−/−) (*P<0.05, **P<0.01, and ***P<0.001) and treatment (†P<0.05, ††P<0.01, and †††P<0.001).