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. 2019 Aug 8;10(8):599. doi: 10.1038/s41419-019-1835-3

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — ER deficiency promoted TFAP-2-inducible LINC00511 expression by enhancing the occupancy efficiency of TFAP-2 at specific promoter regions. The ER-negative-associated LINC00511, which was mainly located in the nucleus, repressed the expression of CDKN1B via interacting with EZH2 to recruit PRC2 to mediate histone methylation, contributing to the G1/S transition to sustain cell proliferation