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. 2019 Aug 9;38:348. doi: 10.1186/s13046-019-1334-5

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — TET1 suppresses pancreatic cell migration, invasion, and EMT. a and b Wound healing assay for TET1-KO, TET1-OE, TET1-MUT, and NC cell migration at 0, 24, and 48 h (left), calculated data (right), **P < 0.01. c and d Transwell assays revealed that overexpression of wild type but not mutant TET1 inhibited cell migration and invasion, while TET1 knockout accelerated migration and invasion, ** P < 0.01. e and f. Western blot analysis of epithelial markers and mesenchymal markers in BXPC-3 and SW1990 cells