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. 2019 Aug 9;9:11580. doi: 10.1038/s41598-019-48043-1

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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(A) Box plots of the trifucosylation degrees of serum AGP and serum AFP levels among patients with cirrhosis, early-stage HCC, and late-stage HCC. The box refers to the 10th and 90th percentile values with a line indicating the median value. Points outside the interquartile range are outliers. The Kruskal-Wallis test showed a significant difference in the trifucosylation degree of AGP and serum AFP level during disease progression from cirrhosis to late HCC (P < 0.0001). (B) Scatter plot of the trifucosylation degree of AGP N-glycans in cirrhosis and HCC patients induced by NASH, ALC, and HCV, respectively. (C–E) Receiver operating characteristics (ROC) curves of the trifucosylation degree to differentiate HCC from cirrhosis cases induced by NASH (C), ALC (D), and HCV (E) respectively.