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. 2019 Aug 5;12:6145–6156. doi: 10.2147/OTT.S209784

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© 2019 Zheng et al.

This work is published and licensed by Dove Medical Press Limited. The full terms of this license are available at https://www.dovepress.com/terms.php and incorporate the Creative Commons Attribution – Non Commercial (unported, v3.0) License (http://creativecommons.org/licenses/by-nc/3.0/). By accessing the work you hereby accept the Terms. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed. For permission for commercial use of this work, please see paragraphs 4.2 and 5 of our Terms (https://www.dovepress.com/terms.php).

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LINC00673 promoted the migration and invasion of EOC cells.

Notes: (A) Relative LINC00673 expression in EOC cell lines compared to HOSEpiC. (B) Knockdown of LINC00673 by siRNAs in OVCAR3 and HO8910. (C and D) LINC00673 silencing inhibited the migration (C) and invasion (D) of OVCAR3 and HO8910 cells. (E) Overexpression of LINC00673 promoted the migration and invasion of A2780 cells. *P<0.05; **P<0.01; ***P<0.001; ****P<0.0001.

Abbreviations: EOC, epithelial ovarian cancer; NC, LINC00673-negative control lentivirus cells; OE, LINC00673-overexpression lentivirus cells; Vector, LINC00673-vector lentivirus cells.