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. 2019 Aug 6;13:2745–2757. doi: 10.2147/DDDT.S213910

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© 2019 Zheng et al.

This work is published and licensed by Dove Medical Press Limited. The full terms of this license are available at https://www.dovepress.com/terms.php and incorporate the Creative Commons Attribution – Non Commercial (unported, v3.0) License (http://creativecommons.org/licenses/by-nc/3.0/). By accessing the work you hereby accept the Terms. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed. For permission for commercial use of this work, please see paragraphs 4.2 and 5 of our Terms (https://www.dovepress.com/terms.php).

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Effects of liraglutide and telmisartan on cardiac hemodynamics after AAC. (A) Representative tracing of the invasive cardiac hemodynamics recording among the different groups. MAP, HR, and left ventricular end-diastolic pressure (LVEDP) were obtained using a pressure transducer inserted into the left ventricle through the right carotid artery. (B) Left ventricular systolic pressure (LVSP) and ±dp/dt_max values of the first derivative of left ventricular pressure were obtained using a pressure transducer inserted into the left ventricle. Values are mean±SEM (n=6/group). *p<0.05 AAC vs Sham; ^#p<0.05 Lira or Telmi vs AAC.

Abbreviations: AAC, abdominal aortic constriction; HR, heart rate.