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. 2019 Jul 23;20(3):2511–2518. doi: 10.3892/mmr.2019.10520

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Copyright: © Wang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — LPS regulates the expression levels of p-S6K, p-AMPK and Sestrin2 in rats. (A) p-S6K, S6K, p-AMPK, AMPK and Sestrin2 protein levels were detected by western blot analysis. (B) Protein levels of p-S6K were adaptively decreased, and those of (C) p-AMPK (D) and Sestrin2 were adaptively increased by LPS treatment, and the change at 6 h was the most significant. (E) The ratio of p-S6K/S6K protein was significantly decreased. (F) The ratio of p-AMPK/AMPK was adaptively increased. **P<0.01 vs. the control group. ^#P<0.05 and ^##P<0.01 vs. the 6 h group, ^{^^}P<0.01 vs. the 12 h group. LPS, lipopolysaccharide; p-, phosphorylated; AMPK, AMP-activated protein kinase.