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. 2019 Aug 13;8(8):42. doi: 10.1038/s41389-019-0153-z

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a Stable overexpression of miR-876 in the KMCH cell line. b Colony formation ability of KMCH cells stably expressing miR-876. c BCL-XL protein expression levels in KMCH cells expressing miR-876. d Stable expression of miR-876 induces apoptosis. e miR-876 expression suppresses in vivo tumor cell growth. f miR-876 suppresses BCL-XL expression in tumor in vivo. g Western blot analysis showing expression of PARP, caspase 3, and PCNA in miR-876-expressing in vivo tumor samples. *p < 0.05