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. Author manuscript; available in PMC: 2020 Aug 15.
Published in final edited form as: Clin Cancer Res. 2019 May 20;25(16):4933–4944. doi: 10.1158/1078-0432.CCR-19-0183

Table 2:

Individual patient responses

Patient information DL Time to progression
(weeks)
Best clinical response
(Δ target lesions from baseline)
KRAS mutation
MSS colorectal cancer 1 12 PR* at 6 wk (−38%)
MSS colorectal cancer 1 26 SD at 12 wk (−4%)
Appendiceal cancer 3 6 PD at 6 wk (+6% + new)
Pancreatic cancer 3 18 SD at 6 wk (−20%)
Appendiceal cancer on capecitabine + bevacizumab 3 80+ SD at 12 wk (−8%)
MSS colorectal cancer 3 82+ PR at 82 wk (−30.6%)
KRAS wildtype
MSS colorectal cancer 2 6 PD at 6 wk (+23%)
MSS colorectal cancer 2 6 PD at 6 wk (+7% + new)
MSS colorectal cancer 2 18 SD at 6 wk (+11%)
MSS colorectal cancer 3 6 PD at 6 wk (+27%)
KRAS unknown or not tested
Endometrial cancer 1 6 PD at 6 wk (+13%)
Salivary gland cancer 3 12+ SD at 12 wk (−2%)**

PR*, unconfirmed partial response; + ongoing treatment on trial; DL, dose level (DL1 = 1 MVA-BN-CV301 injection, DL2 = 2 MVA-BN-CV301 injections, DL3 = 4 MVA-BN-CV301 injections); MSS, microsatellite stable; SD, stable disease; PD, progressive disease; PR, partial response; wk, week; new, new lesion; **tumor resected at 13 weeks on trial. Patient remained on trial with no measurable disease but at high risk of recurrence.