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. 2019 Aug 6;14(10):1233–1236. doi: 10.1016/j.radcr.2019.07.010

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© 2019 The Authors

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig 1 — MRI evidence of resolution of radiation related change post-MRgFUS. All images are presented in radiological orientation. (A) Pre-SRS T2-FLAIR and (B) T1-weighted MR images show no evidence of inflammation in the thalamus. (C) The pre-MRgFUS (post-SRS) T2-FLAIR image shows evidence of SRS-related hyperintensity (white arrow). (D) The pre-MRgFUS (post-SRS) T1-weighted image shows evidence of SRS-related hypointensity (white arrow). (E) One day post-MRgFUS, the T2-FLAIR image shows apparent enhancement of the SRS-related hyperintensity (white arrow), as well as a typical-appearing MRgFUS lesion (black arrow). (F) One day post-MRgFUS, the T1-weighted image shows apparent enhancement of the SRS-related hypointensity (white arrow), as well as a typical-appearing MRgFUS lesion (black arrow). (G) Three months post-MRgFUS, the T2-FLAIR image shows the SRS-related hyperintensity is resolved; a typical-appearing MRgFUS lesion remains (black arrow). (H) Three months post-MRgFUS, the T1-weighted image shows the SRS-related hypointensity is resolved; a typical-appearing MRgFUS lesion remains (black arrow). The hyperintense signal in the basal ganglia visible on the T1-weighted images is thought to be related to the patient's history of cirrhosis.