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. 2019 Aug 19;9:11995. doi: 10.1038/s41598-019-48402-y

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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LDL-C serum level according to the mutation status of the genes LDLR, APOB, and PCSK9. LDL-C serum concentrations (mg/dl) are shown for a population based cohort (BASE-II, N = 1631)). LDL-C serum concentrations of the hypercholesterolemic patients with respect to the mutation status in one of the three FH genes LDLR, APOB, PCSK9 (mutation negative (N = 75) vs. mutation found (N = 68)). Black lines indicate medians and dots single values. One-way ANOVA revealed significant differences between groups (p < 0.001). Post hoc Tukey’s test revealed significant differences between all possible pairs with ***indicating p < 0.001.