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. 2019 Aug 8;9(8):e025884. doi: 10.1136/bmjopen-2018-025884

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© Author(s) (or their employer(s)) 2019. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ.

This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited, appropriate credit is given, any changes made indicated, and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/.

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H₂O₂ production (A), 8-iso-PGF2α formation (B), H₂O₂ breakdown activity (HBA) (C) and NO bioavailability (D) at baseline, after 2 hours (T2h) and at the 5 days (T5d) from the PCI in patients received GSH (n=25, dashed line) or placebo (n=25, continuous line). Data are expressed as mean±SEM (*P<0.05, **P<0.01, ***P<0.001). GSH, glutathione; H₂O₂, hydrogen peroxide; NO, nitric oxide; PCI, percutaneous coronary intervention; PGF2α, prostaglandin F2α.