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. Author manuscript; available in PMC: 2019 Aug 21.
Published in final edited form as: Rheumatology (Oxford). 2017 Dec 1;56(12):2145–2153. doi: 10.1093/rheumatology/kex340

Figure 3. Kaplan-Meier One Minus Survival plots with (A) ACPA-levels in ACPA-positive CSA patients and (B) RF-levels in RF-positive CSA-patients and associated risks for progression to clinical arthritis over time.

Figure 3

A. The ACPA-positive patients in the second tertile (levels 96–325 U/ml, N=11) had a HR of 1.2 for progression to clinical arthritis (95%CI=0.39–3.9) compared to the patients in the lowest tertile (N=10). The ACPA-positive patients in the third and highest tertile (levels ≥326 U/ml, N=11) had a HR of 1.6 for progression to clinical arthritis (95%CI=0.50–4.8) compared to the patients in the lowest ACPA-level tertile.

B. The RF-positive patients in the second tertile (levels 11-40 IU/ml, N=17) had a HR of 1.6 for progression to clinical arthritis (95%CI=0.50–5.0) compared to the patients in the lowest tertile (N=17). The RF-positive patients in the third and highest tertile (levels ≥41 IU/ml, N=17) had a HR of 3.3 for progression to clinical arthritis (95%CI=1.1–9.6) compared to the patients in the lowest RF-level tertile.