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. 2019 Jun 4;33(9):9762–9774. doi: 10.1096/fj.201802519R

Figure 7.

Figure 7

Kcne2 deletion impairs pulmonary protein phosphorylation post I/R. Left: representative Western blots of pERK1/2 and tERK1/2 (A), pp38 MAPK and tp38 MAPK (B), pJNK and tJNK (C), pAKT and tAKT (D), pGSK3β and tGSK3β (E), and pSTAT-3 and tSTAT-3 (F) in Kcne2+/+ and Kcne2−/− lungs post I/R injury; 1 mouse/lane. Right: mean ratio of pERK:tERK (n = 7–8) (A), pp38 MAPK:tp38 MAPK (n = 5) (B), pJNK/tJNK (n = 5–6) (C), pAKT/tAKT (n = 5–6) (D), pGSK3β/tGSK3β (n = 5–9) (E), and pSTAT-3/tSTAT-3 (n = 5) (F) band densities from blots as in left. *P < 0.05, **P < 0.01, ***P < 0.001 compared with sham-treated Kcne2+/+ mice; #P < 0.05, ##P < 0.01, ###P < 0.001 compared with sham-treated Kcne2−/− mice; †P < 0.05, ††P < 0.01, †††P < 0.001 compared with Kcne2+/+ mice post I/R (by 1-way ANOVA).