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. Author manuscript; available in PMC: 2019 Aug 22.
Published in final edited form as: Clin Genet. 2005 Mar;67(3):230–239. doi: 10.1111/j.1399-0004.2004.00392.x

Fig. 2.

Fig. 2.

Scatter plot and correlation data for insulin resistance [(homeostasis model assessment (HOMA)] and triglycerides, cholesterol, and leptin for both obese subjects with Prader–Willi syndrome (PWS) (solid line) and obese controls (dashed line). Obesity was determined for each subject using body mass index (BMI). For adult subjects (≥18 years of age), obesity was defined as BMI ≥ 30. For subjects less than 18 years, obesity was defined as BMI > 95th percentile using published standardized growth charts for each sex (24).