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. 2019 Aug 22;18:126. doi: 10.1186/s12943-019-1054-7

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 8 — CircNHSL1 promotes growth and metastasis of gastric cancer cells in vivo. a Images of subcutaneous xenograft tumors of MKN-28 cells. b Tumor weight of MKN-28 cells was shown. c Tumor volumes of MKN-28 cells measured every 3 days for 6 times were analyzed. d Image of subcutaneous xenograft tumors of SGC-7901 cells. e Tumor weight of SGC-7901 cells was shown. f Tumor volumes of SGC-7901 cells measured every 3 days for 6 times were analyzed. g Representative images of liver metastasis assay and HE staining of the liver of MKN-28 cells. Knockdown of circNHSL1 decreased the number of liver surface metastasis compared with negative control. h Representative images of liver metastasis assay and HE staining of the liver of SGC-7901 cells. Overexpression of circNHSL1 increased the number of liver surface metastasis compared with negative control. i-j Representative images of peritoneal metastasis assays. Knockdown of circNHSL1 decreased the number of peritoneal metastasis nodules compared with negative control (i), whereas overexpression of circNHSL1 increased the number of peritoneal metastasis nodules (j). **p < 0.01