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. 2019 Aug 19;12:6623–6638. doi: 10.2147/OTT.S196630

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© 2019 Wu et al.

This work is published and licensed by Dove Medical Press Limited. The full terms of this license are available at https://www.dovepress.com/terms.php and incorporate the Creative Commons Attribution – Non Commercial (unported, v3.0) License (http://creativecommons.org/licenses/by-nc/3.0/). By accessing the work you hereby accept the Terms. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed. For permission for commercial use of this work, please see paragraphs 4.2 and 5 of our Terms (https://www.dovepress.com/terms.php).

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miR-139-5p was a target of SNHG3. (A) The relationship between SNHG3 and miR-139-5p was predicted by starbase. (B and C) SNHG3-WT or SNHG3-MUT was cotransfected into Hep3B (B) and HepG2 (C) cells with miR-139-3p or their negative controls. (D and E) SNHG3-WT or SNHG3-MUT was cotransfected into Hep3B (D) and HepG2 (E) cells with miR-139-3p inhibitor or their negative controls. (F) SNHG3 overexpression in Hep3B and HepG2 cells. (G) miR-139-5p expression in pcDNA, pcDNA-SNHG3, siNC, and siSNHG3. *P<0.05.

Abbreviation: SNHG3, small nucleolar RNA host gene 3.