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. 2019 Aug 26;7:223. doi: 10.1186/s40425-019-0693-y

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 8 — Anti-sMIC therapy with B10G5 up-regulates PD-L1 expression on tumor cells, which is in part NK cell dependent. a, Depletion of NK cells (dpNK) during therapy diminishes the therapeutic effect of co-targeting sMIC and PD-L1 as assessed by prostate weight at necropsy. All treatments were given twice weekly i.p. for 8 weeks. b, Representative histograms from flow cytometry analyses demonstrating the percentage of PD-L1⁺ tumor cells from TRAMP/MICB mice. c, Summary data of the percentage of PD-L1⁺ tumor cells from (a)