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. 2019 Aug 22;12:6857–6863. doi: 10.2147/OTT.S217473

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© 2019 Chu et al.

This work is published and licensed by Dove Medical Press Limited. The full terms of this license are available at https://www.dovepress.com/terms.php and incorporate the Creative Commons Attribution – Non Commercial (unported, v3.0) License (http://creativecommons.org/licenses/by-nc/3.0/). By accessing the work you hereby accept the Terms. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed. For permission for commercial use of this work, please see paragraphs 4.2 and 5 of our Terms (https://www.dovepress.com/terms.php).

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ELF3-AS1 positively regulated OSCC cell proliferation through GLUT1. Cell proliferation data analyzed by ANOVA (one-way) and Tukey test showed that ELF3-AS1 and GLUT1 overexpression resulted in increased proliferation rate of OSCC cells. However, ELF3-AS1 and GLUT1 siRNA silencing resulted in decreased proliferation rate of OSCC cells. In addition, GLUT1 siRNA silencing attenuated the effects of ELF3-AS1 overexpression. *p<0.05.

Abbreviations: ELF3-AS1, E74 like ETS transcription factor 3-antisense RNA 1; GLUT1, glucose transporter 1.