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. 2019 Aug 28;25:43. doi: 10.1186/s10020-019-0107-0

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© The Author(s) 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 8 — Melatonin improves oxidation in osteoporosis (OP) mice. Oxidative stress-related liver superoxide dismutase (SOD) (a), malondialdehyde (MDA) (b), and glutathione peroxidase (GPX) (c); left kidney SOD (d), MDA (e), and GPX (f); and right kidney SOD (g), MDA (h), and GPX (i) were determined after the administration of melatonin or alendronate for 2 months. Control: normal mice, Model: retinoic acid (RA)-induced OP model mice, Mlt-L: low-dose melatonin-treated OP model mice, Mlt-H: high-dose melatonin-treated OP model mice. Positive control: alendronate-treated OP mice. *, P < 0.05 vs control. #, P < 0.05 vs model; n = 6 per group