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. 2019 Aug 28;17:295. doi: 10.1186/s12967-019-2044-7

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© The Author(s) 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — Proliferation of K14-positive cells in radiation skin-injury mouse model. a Ki-67 positivity (arrows) on immunostain; and b Western blot analysis of p-AKT, AKT, and Ki-67 expression levels at wound sites. Results of one experiment performed in triplicate and expressed as mean ± SEM *p < 0.05 vs. IR group. IR irradiation, CTL control, PRP platelet-rich plasma