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. 2019 Jul 2;23(9):6173–6181. doi: 10.1111/jcmm.14499

Table 4.

Stratification analysis of TUG1 polymorphisms and clinical characteristics of IS

Variables rs2240183 rs5749201 rs5753409 rs7284767 rs8139350
TT CT/CC P TT AT/AA P GG AG/AA P AA AG/GG P CC CG/GG P
TC, mmol/L 4.80 ± 1.05 4.78 ± 1.00 0.78 4.88 ± 0.92 4.70 ± 1.08 0.03 4.81 ± 1.03 4.69 ± 0.97 0.22 4.79 ± 1.01 4.77 ± 1.03 0.82 4.78 ± 1.01 4.80 ± 1.03 0.84
TG, mmol/L 1.94 ± 1.39 1.86 ± 1.44 0.54 1.86 ± 1.26 1.89 ± 1.55 0.81 1.85 ± 1.28 1.97 ± 1.83 0.41 1.94 ± 1.62 1.79 ± 1.07 0.22 1.89 ± 1.48 1.83 ± 1.16 0.71
HDL‐C, mmol/L 1.27 ± 0.41 1.31 ± 0.40 0.31 1.35 ± 0.42 1.25 ± 0.39 0.003 1.30 ± 0.41 1.27 ± 0.39 0.48 1.29 ± 0.40 1.30 ± 0.41 0.70 1.28 ± 0.40 1.34 ± 0.44 0.18
LDL‐C, mmol/L 2.91 ± 0.97 2.79 ± 0.99 0.17 2.80 ± 1.04 2.86 ± 0.94 0.45 2.84 ± 0.98 2.80 ± 0.99 0.66 2.82 ± 1.02 2.85 ± 0.93 0.76 2.84 ± 0.97 2.79 ± 1.04 0.66
VLDL‐C,mmol/L 0.90 ± 0.78 0.80 ± 0.59 0.11 0.82 ± 0.65 0.85 ± 0.67 0.69 0.83 ± 0.62 0.84 ± 0.79 0.94 0.87 ± 0.80 0.79 ± 0.37 0.14 0.85 ± 0.70 0.76 ± 0.43 0.23
Apo‐A1, g/L 1.22 ± 0.24 1.23 ± 0.27 0.73 1.26 ± 0.26 1.20 ± 0.26 0.007 1.23 ± 0.26 1.21 ± 0.24 0.44 1.24 ± 0.26 1.21 ± 0.27 0.23 1.22 ± 0.26 1.25 ± 0.25 0.35
Apo‐B, g/L 1.00 ± 0.30 1.00 ± 0.30 0.97 1.02 ± 0.31 0.99 ± 0.29 0.20 1.00 ± 0.30 1.00 ± 0.31 0.86 1.01 ± 0.31 1.00 ± 0.29 0.83 1.00 ± 0.30 1.01 ± 0.28 0.80
Hcy, μmol/L 14.08 ± 5.04 14.28 ± 6.16 0.70 14.58 ± 6.14 13.91 ± 5.53 0.17 14.33 ± 6.14 13.81 ± 4.55 0.38 14.08 ± 5.84 14.41 ± 5.79 0.50 14.27 ± 6.03 13.96 ± 4.72 0.63
NEFA, mmol/L 0.57 ± 0.29 0.54 ± 0.28 0.14 0.55 ± 0.28 0.55 ± 0.29 0.90 0.55 ± 0.29 0.53 ± 0.28 0.40 0.54 ± 0.28 0.55 ± 0.28 0.61 0.55 ± 0.27 0.52 ± 0.32 0.31

Apo‐A1, apolipoprotein A1; Apo‐B, apolipoprotein B; Hcy, homocysteic acid; HDL‐C, high‐density lipoprotein cholesterol; IS, ischaemic stroke; LDL‐C, low‐density lipoprotein cholesterol; NEFA, non‐esterified fatty acid; TC, total cholesterol; TG, triglyceride; TUG1, taurine‐upregulated gene 1; VLDL‐C, very low‐density lipoprotein cholesterol.