Skip to main content
. Author manuscript; available in PMC: 2020 Jan 1.
Published in final edited form as: Neurochem Res. 2018 Mar 14;44(1):234–246. doi: 10.1007/s11064-018-2502-2

Fig. 1:

Fig. 1:

Balancing survival and function. Neurotoxic factors elevated by HIV act via various upstream mechanisms to converge on the potentiation of NMDARs. Neurons and networks then adapt to the presence of this stimulus by scaling synapses to attenuate Ca2+-mediated toxicity.