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. 2019 Aug 31;16:175. doi: 10.1186/s12974-019-1557-6

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — Neutrophil functions played vital role in the protective effects of atRA treatment. a Schematic diagram showing the time course of neutrophil depletion, atRA administration and tMCAO. b Frequency of neutrophil in peripheral blood was analyzed with flow cytometry to demonstrate the depletion efficacy of anti-Ly6G antibodies. Percentage of CD11b⁺CD45^hiLy6G⁺ neutrophil among singlets were displayed. N = 3 mice per group. ***P ≤ 0.001, versus PBS-treated group in t test. c Infarct volume of mice was quantified with TTC (red)-stained coronal sections (N = 6 mice per group). ***P ≤ 0.001 in one-way ANOVA. d Neurological deficit score was assessed right after reperfusion and 1d after tMCAO (N = 6 mice per group). **P ≤ 0.01 in two-way ANOVA