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. 2019 Jul 30;42(4):1283–1294. doi: 10.3892/or.2019.7254

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Copyright: © Niu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 4. — In vitro activities of CPXM2 in GC cells. AGS and HGC-27 cells were infected with lentiviruses carrying shCPXM2-2, shCPXM2-3, or scrambled control shRNA, and then underwent CCK-8 assay (A and B), colony formation assay (C and D), scratch wound-healing assay (E and F), and Transwell^® migration assay (G and H). CPXM2 silencing in these cells significantly inhibited cell proliferation and migration. Three replicates were conducted for each experiment. *P<0.05, **P<0.01, ***P<0.001 vs. the control group. CCK-8, Cell Counting Kit-8; OD, optical density; CPXM2, carboxypeptidase X, M14 family member 2; GC, gastric cancer.