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. 2019 Aug 30;15:1744806919872602. doi: 10.1177/1744806919872602

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© The Author(s) 2019

Creative Commons Non Commercial CC BY-NC: This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (http://www.creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 6. — Evaluation of blood-brain barrier permeability after inferior alveolar nerve injury in rats. (a) High levels of extravasated Evans blue dye were evident on POD 5 in the inferior alveolar nerve injury group compared to the sham-treated group. There were eight animals in each group. *P < 0.05, sham vs. nerve injury group. (b and c) Western blot analysis revealed significantly decreased occludin and ZO-1 expression in the medullary dorsal horn on POD 5. Inferior alveolar nerve injury did not alter the claudin-5 expression level. GAPDH was used as a loading control. There were eight animals in each group. *P < .05, sham vs. nerve injury group. GAPDH: glyceraldehyde 3-phosphate dehydrogenase.