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. 2019 Sep 4;7:126. doi: 10.1186/s40478-019-0771-x

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — Diffusion of β-amyloid deposits and tau inclusions in Alzheimer-inoculated mouse lemurs. Immunostaining of Aβ (4G8, a-e, k-o (magnified views)) and tau (AT8, f-j, p-y (magnified views)) in 5 successive brain sections. u-y displays magnification of the tau-positive lesions from (f-j) or (p-t). β-amyloid and tau deposits (a-t) were seen exactly at the same locations (arrows). They spread from the inoculation site (open arrow in b, c) to regions localized one millimeter ahead and behind the inoculation site (A1.50 mm to P0.50 mm correspond to spatial references in the Bons atlas [31]). Scale bars: 1 mm (a-j), 200 μm (k-t), 50 μm (u-y)