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. 2005 Feb 23;25(8):2070–2080. doi: 10.1523/JNEUROSCI.4163-04.2005

Figure 2.

Figure 2.

The morphology of motor, hippocampal, and cerebellar cortices was not altered by anisomycin injection. a-f, NeuN immunostaining of the motor, hippocampal, and cerebellar cortices from saline-injected (a-c) and anisomycin-injected (d-f) animals (magnification, 5×). g-j, GFAP immunostaining of the hippocampus obtained from saline-injected (g, h) and anisomycin-injected (i, j) mice. As illustrated at 20× magnification, there was no glial reaction in the CA3 layer in saline-injected (h) or anisomycin-injected (j) animals. k-n, Calbindin immunostaining of the hippocampal and cerebellar cortices of saline-injected (k, l) and anisomycin-injected (m, n) mice. The lack of anisomycin-induced changes in the mossy fiber tract is evidenced by the absence of any noticeable difference in the staining of the stratum lucidum (CA3 field) between saline-treated (k) and anisomycin-treated (m) mice (magnification, 20×). Note the intactness of the Purkinje cell dendritic arborization in the molecular layer of the cerebellum after anisomycin treatment (n) compared with saline-treated animals (l). CA1, CA1 field; CA3, CA3 field; DG, dentate gyrus; PCL, Purkinje cell layer; MF, mossy fiber tract. In all cases, n = 3 per group.