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. 2019 Sep 6;13:289. doi: 10.1186/s13256-019-2197-2

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — a A cross-sectional view of the tumor. Pre-treatment necrosis and hemorrhage is recognized in the center of tumor (asterisk). The mass to the medial side of the main tumor is a benign fibroadenoma (arrow). b On histological examination, nuclear atypia and tight fascicular proliferation of highly cellular spindle tumor cells in the hypercellular areas (high-power view). It also shows the necrotic fibrosis and the weakly stained nucleus known as ghost cells caused by neoadjuvant chemotherapy in hypocellular areas (arrow). c The neoplastic cells are reactive for alpha smooth muscle actin on immunohistochemistry. d The Ki-67 labelling index was approximately 20%. e Necrotic tissue is replaced by granulation and fibrous connective tissue (high-power view)