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. 2019 Sep 6;20:351. doi: 10.1186/s12882-019-1534-4

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — (a) Comparisons of plasma advanced glycation end products (AGEs) levels between groups and treatments. ^* Before versus. After treatments, p < 0.05; ^# any groups compared with the control group before treatment, p < 0.05; ^$ any groups compared with the control group after treatment, p < 0.05; (b) Comparisons of differences of Acetylcholine (Ach)-related vasorelaxation between groups. ^* any groups compared with the control group (Group 1), p < 0.05; ^# any groups compared with the CKD group (Group 2), p < 0.05. (c) Comparisons of inhibitory effect of N(omega)-Monomethyl-L-Arginine Acetate (L-NMMA) on the Ach-related vasorelaxation between groups. (d) Comparisons of sodium nitroprusside (SNP)-related vasorelaxation between groups