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. 2019 Sep 6;20:351. doi: 10.1186/s12882-019-1534-4

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — Results derived from the immunohistochemical stain of thoracic aortas obtained from the Sprague-Dawley rats. (a) Accumulation of the advanced glycation end products (AGEs) in thoracic aorta tissue was higher in the chronic kidney disease (CKD) group compared with it was in the control, CKD + ALT-711, and CKD + spironolactone groups. (b) Amount of phospho-endothelial nitric oxide synthase (p-eNOS) in the thoracic aorta tissue was lower in the CKD group compared with it was in the control, CKD + ALT-711, and CKD + Spironolactone groups