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. 2019 Sep 3;10:2095. doi: 10.3389/fimmu.2019.02095

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Copyright © 2019 Park, Kwon, Lee, Kwon, Ko, Kim and Kim.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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The role of Fpr2 in WKYMVm-induced immune resolution in scleroderma. (A,B) Wm-induced inhibition of systemic inflammation in scleroderma. The BLM-induced scleroderma mice model were treated with or without Wm for 3 weeks, and the serum levels of TNF-α (A) and INF-γ (B) in the mock-treated (PBS) and the scleroderma mice (BLM or BLM+Wm) were determined by ELISA assay. (C,D) The role of Fpr2 in Wm-induced suppression of systemic inflammation in scleroderma. The BLM-induced scleroderma model of wild type and Fpr2 KO mice were daily injected with or without Wm for 3 weeks, and the serum levels of TNF-α (C), and INF-γ (D) in the scleroderma mice were quantified by ELISA analysis. Data represent mean ± SD (n = 8 per group). *p < 0.05.