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. 2019 Sep 9;10(9):647. doi: 10.1038/s41419-019-1876-7

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a The expression of Smad1, 5, and their phosphorylation levels were measured with the upregulation or downregulation of LMCD1. b, c Relative BRE Luciferase activity was measured with the upregulation or downregulation of LMCD1 in 293T cells. d ALP staining and AR staining showed that the restricted osteogenic capacity of LMCD1-downregulated BMSCs was rescued by BMP2 treatment. e ALP staining and AR staining showed that the enhanced osteogenic ability of LMCD1-upregulated BMSCs was reduced by Noggin treatment