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. 2019 Sep 4;10:1140. doi: 10.3389/fphys.2019.01140

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Copyright © 2019 Wang, Liu, Lian, Shentu, Fang, Shao, Chen, Wang, Zhou and Sun.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Branched-chain amino acid (BCAA) catabolic defect reduces body weight with beneficial effects on glucose metabolism in PP2Cm KO mice. Body weight (A, n = 10–11), food intake (B, n = 10–11), respiratory exchange ratios (RER) (C, n = 9/genotype), average RER during light and dark cycles (D, n = 9/genotype), glucose tolerance test (E,G) and insulin tolerance test (F,H) of WT and PP2Cm KO male mice fed a normal diet (n = 8 for each group). Data are represented as means ± SEM. ^∗p < 0.05; ^∗∗p < 0.01; ^∗∗∗p < 0.001 compared to WT.