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. 2019 Sep 12;16(9):e1002901. doi: 10.1371/journal.pmed.1002901

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© 2019 Ruscitti et al

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig 2 — Metabolic measures of glycaemic control at baseline (T0) and after 3 (T3) and 6 (T6) months of treatment with anakinra or TNFi. (A) HbA1c%. T0: anakinra group (7.73% ± 0.67) versus TNFi group (7.83% ± 0.76); T3: anakinra group (6.95% ± 0.61) versus TNFi group (7.63% ± 0.68), p = 0.0038; T6: anakinra group (6.70% ± 0.67) versus TNFi group (7.64% ± 0.65), p < 0.001. (B) FPG. T0: anakinra group (139.05 ± 50.09 mg/dL) versus TNFi group (139.25 ± 29.55 mg/dL); T3: anakinra group (109.78 ± 30.58 mg/dL) versus TNFi group (133.06 ± 27.72 mg/dL), p = 0.027; T6: anakinra group (100.81 ± 11.11 mg/dL) versus TNFi group (140.93 ± 39.45 mg/dL), p < 0.001. FPG, fasting plasma glucose; HbA1c%, percentage of glycated haemoglobin; TNFi, tumour necrosis factor inhibitor.