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. Author manuscript; available in PMC: 2020 Sep 13.
Published before final editing as: J Am Acad Child Adolesc Psychiatry. 2019 Mar 13:S0890-8567(19)30197-2. doi: 10.1016/j.jaac.2019.03.013

Figure 1: Biometric cross-lagged reduced model for Callous-Unemotional Behavior and negative parenting, shown for one twin.

Figure 1:

Dashed paths indicate nonsignificant paths that could be dropped from the model. The model constrains all cross-age associations to function as phenotypic partial regression coefficients. The paths leading from CU at age 2 to CU at age 3 index stability, controlling for any prior association with parenting. The cross-lagged paths reflect the effects of CU at age 2 on parenting at age 3 and visa versa, above and beyond their preexisting relation at age 2 and stability effects. The variances of CU and parenting at age 2 are decomposed into their genetic (A), shared environmental (C), and nonshared environmental (E) components. The genetic and environmental effects at age 3 are residual effects that are independent of age 2, and reflect change. Paths between the latent A, C, and E factors and each phenotype are standardized partial regression coefficients, and indicate the relative contributions of A, C, and E factors to each phenotype. The square of these paths represents the genetic and environmental variance associated with each phenotype. Confidence intervals are presented in parentheses. rg, rc, and re denote the genetic and environmental correlations. A=Genetic influences; C=Shared environmental influences; E=Nonshared environmental influences; CU=Callous-Unemotional BehaviorsaResidualized for sex and conduct problems.